Used in the treatment of acute decompensated heart failure, it improves cardiac contractility without increasing myocardial oxygen demand. It works by enhancing the sensitivity of cardiac troponin C to calcium, leading to improved systolic function. Additionally, it opens ATP-sensitive potassium channels in vascular smooth muscle, resulting in vasodilation and reduced cardiac preload and afterload. This dual action supports heart function and improves symptoms in patients with compromised cardiac output. It is typically administered intravenously in hospital settings, especially when other therapies are insufficient.